By providing a number of Core Facilities, the NORC facilitates the integration and coordination of the abundant ongoing activities throughout the UW system. Our overarching goals are to foster new interdisciplinary research collaborations, stimulate new research activities, improve nutrition and obesity education at multiple levels, and facilitate optimal nutritional management of patients.

Core facilities are available for use by Affiliate Investigators, who are broadly dispersed throughout several schools and numerous departments and divisions within the University of Washington.

The University of Washington NORC is supported by the National Institutes of Health, NIDDK, grant # P30 DK035816. Please cite this grant support in your publications if you have utilized NORC facilities for the research.

Recent Publications

All-trans-retinoic Acid Enhances Mitochondrial Function in Models of Human Liver.

Tripathy S, Chapman JD, Han CY, Hogarth C, Arnold SL, Onken J, Kent T, Goodlett DR, Isoherranen N.  All-trans-retinoic Acid Enhances Mitochondrial Function in Models of Human Liver. Mol Pharmacol 2016. PMCID: PMC4851298

Effects of anxiety on caloric intake and satiety-related brain activation in women and men.

Mestre ZL, Melhorn SJ, Askren MK, Tyagi V, Gatenby C, Young LL, Mehta S, Webb M, Grabowski TJ, Schur EA. Effects of anxiety on caloric intake and satiety-related brain activation in women and men. Psychosom Med 2016. PMCID: PMC4851583

Central injection of fibroblast growth factor 1 induces sustained remission of diabetic hyperglycemia in rodents.

Scarlett JM*, Rojas JM*, Matsen ME, Kaiyala KJ, Stefanovski D, Bergman RN, Nguyen HT, Dorfman MD, Lantier L, Wasserman DH, Mirzadeh Z, Unterman TG, Morton GJ, Schwartz MW. Central injection of fibroblast growth factor 1 induces sustained remission of diabetic hyperglycemia in rodents. Nat Med. In Press, 2016. (*contributed equally to this work)

Parabrachial CGRP neurons control meal termination.

Campos CA, Bowen AJ, Schwartz MW, Palmiter RD. Parabrachial CGRP neurons control meal termination. Cell Metab 23:811-20, 2016. PMCID: PMC4867080

Chronic CNS oxytocin signaling preferentially induces fat loss in high fat diet-fed rats by enhancing satiety responses and increasing lipid utilization.

Blevins JE, Thompson BW, Anekonda VT, Ho JM, Graham JL, Roberts ZS, Hwang BH, Ogimoto K, Wolden-Hanson TH, Nelson J, Kaiyala KJ, Havel PJ, Bales KL, Morton GJ, Schwartz MW, Baskin DG.  Chronic CNS oxytocin signaling preferentially induces fat loss in high fat diet-fed rats by enhancing satiety responses and increasing lipid utilization. Am J Physiol Regul Integr Comp Physiol 2016.  PMCID: PMC4867381

Functional identification of a neurocircuit regulating blood glucose.

Meek TH, Nelson JT, Matsen ME, Dorfman MD, Guyenet SJ, Damian V, Allison MB, Scarlett JM, Nguyen HT, Thaler JP, Olson DP, Myers MG, Jr., Schwartz MW, Morton GJ. Functional identification of a neurocircuit regulating blood glucose. Proc Natl Acad Sci USA 2016. PMCID: PMC4833243

Upcoming Seminars

06/01


Fellows Award Lectures

Fellows Award Lectures